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PATHOPHYSIOLOGY: ACUTE GLOMERULONEPHRITIS

infection somehere in the body immune respoonse dead WBCs general circulation kidney s smaller particles glomeruli excreted via urine #-$ filtration thickening of glomerular basement membrane destruction of glomeruli progresse s affects the %hole kidney reninangiotensin imbalance hypertensio n puffy face C&O' excretion albumin level fluid shifting edema of extremitie s mixed %ith urine hematuri a bleedin g hemoglobi n Oxygen-carrying capacity of the blood Oxygenation of cells and tissues cell metabolism fatigability untreated or prolonged scarring of kidney tissues filtering surface renal failure bigger particles dead M.O. live M.O. general circulation kidney s antige-antibody reaction particle build-up lodged: glomerular basement filtration oliguri a continuous blood circulation pressure

thickening of glomerular basement membrane adds insult ! "

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