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  • Congestive Heart Failure Pathophysiology Schematic Diagram

    Enviado por

    JacinthaVanathayah
    1K visualizações3 páginas
    Heart failure is caused by both modifiable and non-modifiable factors that damage the heart and blood vessels over time. Increased age and being female are non-modifiable risk factors. Modifiable factors include sedentary lifestyle. Damage to blood vessels from plaques and decreased elasticity impair blood flow. The heart works harder to pump blood, causing the muscles to stretch and not contract effectively. This leads to less blood being pumped, activating stress pathways and cardiac remodeling. Vital organs receive inadequate perfusion, impairing their function and leading to edema, fatigue, shortness of breath, and other symptoms of heart failure.

    Descrição original:

    pathophysiology of CHF

    Título original

    Congestive Heart Failure Pathophysiology Schematic Diagram (1)

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    Heart failure is caused by both modifiable and non-modifiable factors that damage the heart and blood vessels over time. Increased age and being female are non-modifiable risk factors. Modifiable factors include sedentary lifestyle. Damage to blood vessels from plaques and decreased elasticity impair blood flow. The heart works harder to pump blood, causing the muscles to stretch and not contract effectively. This leads to less blood being pumped, activating stress pathways and cardiac remodeling. Vital organs receive inadequate perfusion, impairing their function and leading to edema, fatigue, shortness of breath, and other symptoms of heart failure.

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    © All Rights Reserved
    Baixe no formato DOCX, PDF, TXT ou leia online no Scribd
    Sinalizar o conteúdo como inadequado
    1K visualizações3 páginas

    Congestive Heart Failure Pathophysiology Schematic Diagram

    Enviado por

    JacinthaVanathayah
    Heart failure is caused by both modifiable and non-modifiable factors that damage the heart and blood vessels over time. Increased age and being female are non-modifiable risk factors. Modifiable factors include sedentary lifestyle. Damage to blood vessels from plaques and decreased elasticity impair blood flow. The heart works harder to pump blood, causing the muscles to stretch and not contract effectively. This leads to less blood being pumped, activating stress pathways and cardiac remodeling. Vital organs receive inadequate perfusion, impairing their function and leading to edema, fatigue, shortness of breath, and other symptoms of heart failure.

    Direitos autorais:

    © All Rights Reserved
    Baixe no formato DOCX, PDF, TXT ou leia online no Scribd
    Sinalizar o conteúdo como inadequado
    de 3

    PATHOPHYSIOLOGY: (Book -based)

    Modifiable Factors:

    Non-modifiable Factors:

    Increase in age- 55 years old and


    above
    Gender- female

    Sedentary lifestyle

    Decreased elasticity of blood vessels and formation of plaques


    on blood vessels
    Narrowing of the blood vessels
    Necrosis and scarring of the vascular endothelium
    Impediment of blood flow to the body

    Increased workload of the heart


    Dilation of ventricles
    Increased in preload

    Increased stretching of myocardial muscle

    Excessive stretching of myocardial muscle

    Ineffective cardiac muscle contraction and increase O2 demand of


    cardiac muscle cells

    Decreased contraction of cardiac muscle

    Decreased cardiac output and systemic perfusion


    www.NursesLabs.com

    Activation of neurohormonal pathways in order to increase circulating


    blood vessel

    Continued neurohormonal stimulation

    Cardiac remodelling

    Decreased blood filling

    Increased stroke volume and decreased cardiac output

    Increased wall tension

    Inadequate perfusion

    PALLOR

    Decreased
    blood flow to
    the kidneys
    Kidneys
    produce
    hormones
    Salt and water
    retention
    EDEMA

    Decreased
    perfusion in the
    coronary
    arteries
    Deprivation of
    cardiac muscle
    cells of nutrients
    needed for
    survival

    Increase
    pulmonary
    pressure

    Separation of
    mitral valve
    leaflets

    FATIGUE &
    WEAKNESS

    Increase
    pulmonary
    pressure
    Impaired left
    ventricular
    relaxation

    Normal Balance
    between oxygen
    supply and demand
    is disrupted

    Increase diastolic
    pressure exceeding
    hydrostatic &
    osmotic pressure in
    pulmonary
    capillaries

    ISCHEMIA

    Increased
    capillary
    pressure in the

    Conversion of
    aerobic
    metabolism to
    anaerobic

    Causes reduced
    contractility

    Decreases
    the hearts
    ability to

    Fluid shifts from the


    circulating blood
    into the interstitium,
    bronchioles, bronchi
    & alveoli

    Decreased
    adenosine
    phosphate
    Increased lactic
    acid production

    Decreased lung
    expansion

    Pulmonary
    Congestion
    www.NursesLabs.com

    BRADYCARDIA

    Irritation of
    myocardial cells

    CHEST PAIN
    POSITIVE
    TROPONIN T

    DYSPNEA

    Fluid trapped in
    pulmonary
    trees
    BILATERAL
    CRACKLES

    www.NursesLabs.com

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