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In diabetic state, the cells may stop responding to insulin or the pancreas
may stop producing insulin intirely.
CLASSIFICATION OF DIABETES
a. cause
b. clinical course
c. treatment
OVERVIEW
insulin is secreted by beta cells, which are one of four types of cells in the
islets of Langerhans in the pancreas.
Insulin is an anabolic, or storage, hormone.
Insulin secretion increases and moves glucose from the blood into muscle,
liver, and fat cells, when the person eats a meal.
In those cells, insulin:
insulin also inhibits the breakdown of stored glucose, protein, and fat.
• in subsequent pregnancies
• 30%-40% will develop overt
diabetes within 10 years
CLINICAL MANIFESTATION
ASSESSMENT
1. history
2. physical examination
3. laboratory examination
4. need for referral
MANAGEMENT
Goal:
1. Nutritional Management
Goals:
2. Exercise
• Benefits
• Exercise precaution
• Exercise recommendations
♦ Result does not accurately reflect the blood glucose level at the time of the
rest.
♦ The renal threshold foe glucose is 180-200 mg/dL, for above target blood
glucose levels.
♦ Hypoglycemia cannot be detected because a “negative” urine glucose
result may occur when the blood glucose leel ranges from 0-180 mg/dl or
higher.
♦ Patients may have a false of being in good control when results are
always negative.
♦ Various medications may interfere with the test result.
♦ In elderly patients and patients with kidney disease, the renal threshold is
raised, thus the false negative may occur at dangerously elevated glucose
levels.
4. Pharmacology therapy
NURSING MANAGEMENT
1. Education
2. Developing a Diabetes Teaching Plan
o Organizing Information
Teaching Survival Skills
• Outline of survival information include:
o 1. Simple pathophysiology
a. Basic definition of diabetes
b. Normal blood glucose ranges and target blood glucose levels.
c. Effects of insulin and exercise
d. Effects of food and stress, including illness and infections
e. Basic treatment approaches
2. Treatment modalities
a. Administration of insulin and oral antidiabetes medications
b. Diet information
c. Monitoring of blood glucose and ketones
3. Recognition, treatment, and prevention of acutr complications
a. Hypoglycemia
b. Hyperglycemia
4. Pragmatic information
a. Where to buy and store insulin, syringes and glucose monitoring
supplies
b. When and how to reach the physician
Simply the treatment regimen if it is too difficult for the patient to follow.
Adjust the treatment regimen to meet patient’s request.
Establish a specific plan or contract with the patients with simple, measurable
goals.
Provide positive reinforcement of self-care behaviors performed instead of
focusing on behaviors that were neglected.
Help the patients to identify personal motivating factors rather than focusing on
wanting to please the doctors or nurse.
Encourage the patients to pursue life goals and interest: discourage an undue
focus on diabetes.
Continuing Care
Clinical Manifestations
• Mild hypoglycemia
• Blood glucose levels falls
• Sympathetic nervous stystem is stimulated
• Resulting in a surge of epinephrine and norepinephrine
• Causes symptoms sush as sweating, tremor, tachycardia, palpitation, nervouaness
and hunger
• Moderate hypoglycemia
• Fall in blood glucose level deprives the brain cells of needed for functioning.
• Signs of impaired function of the CNS may include inability to concentrate,
headache, lightheadedness, confusion, memory lapses, numbness of the lips and
tongue, slurred speech impaired coordination, emotional changes, irrational or
combative behavior, double vision and drowsiness.
• Severe hypoglycemia
• CNS function is so impaired that the patient needs the assistance of another person
for treatment of hypoglycemia.
• Symptoms may include disoriented behavior, seizures, difficulty arousing from sleep,
or loss of consciousness.
Management
1. Teaching Patients
2. Initiating Emergency Measures
3. Promoting Home and Community-Based Care
2. Diabetic Ketoacidosis
Clinical Manifestation
Prevention
• For patients with type 1 diabetes, inability to retain oral fluids, may warrant
hospitalization to avoid diabetic ketoacidosis and possibly coma.
Medical Management
1. Rehydration
2. Restoring Electrolytes
3. Reserving Acidosis
Clinical Manifestation
♦ hypotension
♦ dehydration ( dry mucous membrane, poor skin turgor)
♦ tachycardia
♦ variable neurologic signs (eg. Alteration of sensorium, seizures, hemiparesis)
♦ laboratory Test
♦ blood glucose
♦ electrolytes
♦ BUN
♦ Complete blood count
♦ Serum osmolality
♦ And arterial blood gas analysis
♦ mental status changes,
Medical Management
♦ fluid replacement
♦ correction of electrolyte imbalance
♦ insulin administration
Nursing Management
Nursing Intervention
4. Improving Self-Care
a. Patients teaching
b. Hypokalemia
>Are seen in both type 1 and type 2 diabetes but usually do not occur with
in 1st 5-10 yrs. Of diagnosis
>Renal disease is more prevalent among patient with type 1 diabetes
>Cardiovascular complication is more prevalent among older patient with
type 2 diabetes
MACROVASCULAR COMPLICATION
MANAGEMENT
Diet and exercise- for managing obesity, hypertension, hyperlipidemia
Use of medication- to control hypertension and hyperlipidemia
Smoking cessation
Control blood glucose- to reduce triglyceride\
Patient may require increase ed the amount of insulin
May need to switch from oral antidiabetic agents to insulin
MEDICAL MANAGEMENT
1. intensive insulin therapy- decreases development of retinopathy
2. Argon laser Photocoagulation- main treatment of diabetic
retinopathy.
a. This is a laser treatment that destroys blood vessel and
areas of neovascularization
3. panretinal photocoagulation- patient increase risk for hemorrhaging
-reduces the rate of progression to blindness
-this stops the widespread of new vessel and
hemorrhaging of damaged blood vessel
4. Vitrectomy- removal of fluid with a special drill like instrument and
replaced with saline
NURSING MANAGEMENT
- regular opthalmoligoc examination
- Blood glucose control
- Self management of eye care regimen
NEPHROPATHY
- Patient with type 1 diabetes frequently show initial
signs of renal disease after 10-15 yrs
- Type 2 diabetesw develop renal disease within 10
yrs of the diagnosis
- Soon after the onset of diabetes and especially if the
glucose levels are elevated, the kidney’s filtration
mechanism is stressed, allowing blood protein to
leak into the urine. Pressure in the blood vessels of
the kidney increases
ASSESSMENT
Albumin is the most important blood proteins that leaks into the
urine
1. urine should be checked annually for microalbuminuria
2. Urine dipstick test for albumin, creatinine and BUN level are
obtained
MANAGEMENT
- Control of hypertension – use of ACE inhibitors
- Prevention or treatment of UTI
- Avoidance of nephroroxic substances
- Adjustment of medication as renal Ffunction
changes
- Low Sodium diet
- Low protein diet
>if the patient has already developed microalbuminuria and level exceeds
30mg/24hours in 2 consecutive test. An ACE inhibitor should be
prescribed
IN chronic endstage of renal failure
1. Hemodialysis
2. Peritoneal dialysis has major risk factor --- infection and
peritonitis
Laser or surgery may be performed
DIABETIC NEUROPATHIES
- Include the peripheral sensorimotor, autonomic and
spinal nerve
- Elevated blood glucose level over a period of years
have been implicated in the etiology of neuropathy
- Capillary basement membrane thickening and
demyelinization of the nerves related to
hyperglycemia
- Nerve conduction is disrupted when there are
aberration of the myelin sheaths
- Most common type of diabetic neuropathy are
1. sensorimotos polyneuropathy commonly
affects the distal portions of the nerves
2. Autonomic neuropathy
3. Cranial nerve also occurs in diabetes common
among elderly
Peripheral Neuropathy
Signs and symptoms
1. paresthesia
2. burning sensation
3. feet become numb
4. decreased sensation of light touch which can lead
to an unsteady gait
5. decreased sensation to pain and temperature
place patient with neuropathy at high risk for foot
injury
6. Chariot joint – neuropathy related to joint.
Abnormal distribution on joint due to lack of
propioception
7. On physical examination the there is a decreased
in deep tendon reflex
MANAGEMENT
- intensive insulin therapy and control of blood
glucose level that delays the progression of
neuropathy
- for some patients neuropathic pain spontaneously
resolve within 6 mos.
- Nonopiod analgesic
- Triclylic antidepressant
- Transcutaneous electric nerve stimulation
AUTONOMIC NEUROPATHY
- THERE ARE THREE MANIFESTATIONS
1. cardia
2. GI
3. renal system
- Cardiovascular symptoms range from fixed, slightly
tachycardic heart rate, orthostatic hypertension,
silent or painless myocardial ischemia and infarction
- Delayed gastric emptying bloating, nausea and
vomiting
- In addition there is unexplained absorption of
glucose from ingested food secondary to the
inconsistent gastric emptying
- Urinary retention a decreased sensation of bladder,
fullness and other urinary symptoms of neurologic
bladder result form autonomic neuropathy
- HYPOGLYCEMIC UNAWARENESS
1. autonomic neuropathy of the adrenal medulla
is responsible for diminished or absent
adrenergic symptoms of hypoglycemia.
a. Patient may report that they no longer
feel the shakiness, sweating,
nervousness and palpitations
associated to hypoglycemia
2. Sudomotor neuropathy
- refers to a decrease or absence of sweating
(adhidrosis) of the extremities
3. Sexual dysfunction
- impotence in men and
- decreased libido, reduced vaginal secretions
in women
- if there is vaginal infection there is vaginal
itching, decreased lubrication and tenderness
-
MANAGEMENT
-
Avoid strenuous activities
-
High sodium diet in orthostatic hypotension
-
The discontinuation of medication that impede
autonomic response and use of sympathomometics
and other agents
- That stimulate an autonomic response and the use
of lower body elastic garments that minimize
venous return and prevent pooling of blob in the
extremities
- Treatment delayed gastric emptying includes low fat
diet, small frequent meals, close blood glucose
control and the use of other agents that increases
gastric motility
- Treatment of diarrhea bulk forming laxative or
antidiarrheal agents
- Treatment for constipation use laxative and enemas
FOOT AND LEG PROBLEMS
- Complications of diabetes that contribute to the
increased risk of foot infection…
1. Neuropath: sensory neuropathy leads to loss of
pain and pressure sensation and autonomic
neuropathy leads to dryness and fissuring of
the skin(secondary to sweating decrease
2. Peripheral vascular dse. Poor circulation of the
lower ext,. contributing to poor wound healing
and development of gangrene
3. immunocompromise hyperglycemia impairs the
ability of specialized leukocytes to destroy
bacteria.
- typical sequence of events in the development of
diabertic foot ulcer begins with a soft tissue injury of
the foot. Formation of a fissure between the toes or
in an area pf dry skin or formation of callus.
- Drainage, swelling redness from cellulitis of the leg
or gangrene may be the 1st sign of foot problems
- Treatment of foot ulcer involves bedrest, antibiotics
and debridement
- In patient with peripheral vascular dse. Ulcers of the
foot may not heal because there is decrease oxygen
- Amputation may be necessary to prevent infection
HIGH RISK PERSONS ARE
1. Duration of diabetes more than 10n years
2. age older than 40 yrs.
3. history of smoking
4. decreased peripheral pulse
5. decreased sensation
6. anatomic deformities
7. history of foot ulcer or amputation
MANAGEMENT
1. the feet must be inspected daily for redness, blisters, fissures,
calluses, ulceration, changes in skin temperature and
development of deformities
Aspect of preventive foot care
- proper bathing, drying, and lubricating the feet
- wearing closed toe shoe that fit well
- trimming of toenails
- reducing risk factors such as smoking, elevated
lipids
- avoiding home remedies to treat foot problems
HYPERGLYCEMIA DURING HOSPITALIZATION
Factors that may contribute to hyperglycemia
1. changes in the usual treatment regimen
2. medications
3. IV dextrose
4. mismatched timing of meals and insulin
- short acting insulin is usually needed to avoid
postprandial hyperglycemia
- IV antibiotics should be mixed in normal saline to
avoid excess infusion of dextrose
HYPOGLYCEMIA DURING HOSPITALIZATION
- Factors that may contribute to hypoglycemia
1. Overuse of regular insulin
2. lack of dosage change when dietary intake is
changed
3. overuse medications for hyperglycemia
- successive does of subcutaneous regular insulin
should be administered no more frequently that
every 3-4 hours
- Should have snacks\