PATHOPHYSIOLOGY

NON MODIFIABLE: MODIFIABLE: UNDERLYING DISEASE CONDITION:

-Presence of Foreign Herniation *Telescoping of volvulus adenocarcinoma
-Family History with bodies ingested(picca) Protrusion of 1 part of intestine twisting of the of the colon
History of Colorectal * Fiber diet intestines through into another intestine tumor growth into
Cancer * OFI weakness of abdominal *INTUSSUSCEPTION occlusion intestinal lumen
* Fat intake Muscle/inguinal ring strangulation of of blood
Blood supply supply

*Abdominal Surgery Complication (anesthesia)

(Exploratory laparotomy, loop colostomy, ileostmomy,
Appendectomy)
Loss of Peristaltic activity
Paralytic ileus
MECHANICAL OBSTRUCTION FUNCTIONAL/ NON-MECHANICAL
In the COLON OBSTRUCTION
 Reabsorption of bicarbonate from impaired H2O accumulation of swallowed *constipation of no
Hydrogen ions from pancreatic secretions & electrolyte air & gas content food passage of stool
Gastric juice & bile that are not reabsorption or no passage of flatus
absorbed

Fluid
* DIARRHEA accumulation

Loss of fluid &

Electrolyte @ the GI collection of fluid & gas
K continuous loss of proximal to the site
Hypokalemia F&E
Fatigue DISTENTIO *(Abdominal girth of 26inches)
N
Weakness severe dehydration
Muscle cramping pressure of the intestine

Hypovolemia plasma volume reflux of gastic * food intake CHON intake

CVP tachycardia hemoconcentration contents glocuneogenesis muscle wasting
Hypovolemic Shock *vomiting * glucose (RBS 60mg/dl) pain at the extremeties
(bile stained) cell starvation poor wound healing
RR HR BP utilization of fats lipolysis ketone formation
 DISTENTION

*colicky abdominal pain alteration in presence of visible

(pain scale: 8/10) peristaltic movement peristalsis

*Guarding *irritability *restlessness borborygmus high-pitched

behavior *facial grimace peristalsis
upon moving
intraluminal pressure

edema of intestinal wall venous compression impaired artetial flow

capillary permeability venous return arterial blood flow
leakage of fluid into lumen cardiac output O2 supply to mocusa
promotes rapid growth * blood pressure (90/60) mucosa wall ischemia mucosal wall
of bacteria mucosal wall injury injury
rapid growth of anaerobic
bacteria
production of lethal endotoxins perforation

entry of endotoxins into blood stream

Entry of endotoxins into blood stream perforation GI bleeding
Spillage of gastric content
To peritoneum melena anemia
peritonitis * RBC( 3.9L)
*muscle wasting
*fatigue
INFECTION *weight loss
From 40kg to 33kg
* WBC (13.3mm) * Body temperature ( 38.6 degrees celcius)