Escolar Documentos
Profissional Documentos
Cultura Documentos
07/18/09 1
Anterior
Tibialis anterior
Extensor digitorum longus
Extensor hallicus
Deep peroneal nerve
Anterior tibial artery and vein
07/18/09 2
Lateral
Peroneus longus
Peroneus brevis
Superficial peroneal nerve
07/18/09 3
Superficial Posterior
Soleus
Gastrocnemius
Tendons of the plantaris
07/18/09 4
Deep Posterior
Flexor digitorum
Flexor hallicus longus
Tibialis posterior
Peroneal and posterior tibial artery and vein
Tibial nerve
07/18/09 5
Compartment Muscles
Anterior
Extensors
Shock absorbers
Lateral
Evertors
Superficial Posterior
Plantar flexors
Deep Posterior
Invertors
Stabilizers overpronation
07/18/09 6
Shin Splints
Tendinitis
Periostitis
Muscle strain
Interosseus membrane strain
07/18/09 7
Types of Shin Splints
Anterior
Tibialis anterior
Extensor hallucis longus
Extensor digitorum longus
Posterior
Tibialis posterior
Flexor hallucis longus
Flexor digitorum longus
07/18/09 8
Presentation
07/18/09 9
Treatment
Acute
Rest
Ice
Support – elastic tape
Support at the foot – taping and/or medial
heel wedges.
Orthotics – recurrent/chronic
Calcium – one study
07/18/09 10
Compartment Syndrome
Presentation
Most common is the athlete/exercise.
Complains of aching or cramping of leg following
exercise.
Pain is relieved by rest initially.
Numbness/paresthesia may be present into parts of the
foot, in the distribution of the corresponding nerve.
Local trauma or fracture may also result in this syndrome.
Fascial defects with muscle herniations are present in
~ 40% of patients.
07/18/09 11
Etiologies for Pressure in
Compartments
Patients who have crush injuries or fractures with marked
swelling are at risk for developing ACS.
Patients who have had reduction of long-bone fractures and
those who have experienced prolonged external pressure
from pneumatic antishock garments, casts, or tight-fitting or
blood-soaked dressings.
ACS occurs when the function of muscles, blood vessels,
and nerves is jeopardized by the pressure within the layers
of semirigid fascia that support and partition them.
07/18/09 12
Pathophysiology of Compartment
Syndrome
After an injury to an extremity, an immediate inflammatory
response results in decreased blood flow distal to the injury
and tissue hypoxia.
Inflammatory mediators are released and cause the capillary
wall to lose integrity and colloid proteins to leach into the
soft tissue, drawing more fluid into the soft tissue.
This fluid shift causes increased edema, and the cycle is
perpetuated.
The imbalance in pressures between inflow of arterial blood
and outflow of venous blood eventually culminates in total
cessation of blood flow into the affected extremity.
07/18/09 13
Compartmental Pressures
Intracompartmental pressures in excess of 30 to 40 mm Hg can cause
muscle ischemia; pressure greater than 55 to 65 mm Hg causes irreversible
muscle death.
Normal pressure within a compartment is 8 mm Hg or less.
Even within four to six hours of the onset of ACS, a patient may develop
irreversible muscular damage.
ACS sequelae can run the gamut from foot drop due to peroneal nerve
injury to something as severe as a completely insensate, nonfunctional
extremity with muscle fibrosis and joint contracture (Volkmann's ischemic
contracture).
Potential systemic complications include myoglobinuria, which can lead to
renal failure, and untreated infection of an extremity, leading to sepsis.
In a worst-case scenario, a patient may require amputation.
07/18/09 14
Evaluation
07/18/09 15
Signs of ACS
07/18/09 16
Pain
Is a crucial, though not specific, early sign.
Red flags include:
Patient's complaint of diffuse pain that's not relieved by
analgesics
Pain that's greater during passive motion rather than
during active motion.
Severe pain that's out of proportion to the injury.
07/18/09 17
Paresthesia
Abnormal sensations such as burning, may be the
first symptom to appear, as nerves are very
sensitive to pressure.
07/18/09 18
Pressure
Refers to tension felt during palpation.
As compartment pressure rises, the affected area
becomes extremely taut and feels firm to the touch.
07/18/09 19
Pallor
Caused by pressure in the compartments that
suffered arterial injury, is a late and ominous sign.
07/18/09 20
Paralysis
Is usually a late symptom.
It is caused by either prolonged nerve compression
or irreversible muscle damage.
07/18/09 21
Pulselessness
Is a late and ominous sign, indicating death of
tissue.
A pulse is present in the limb until the very late
stages.
07/18/09 22
Patient Assessment
Compare the injured limb with the non-injured limb at least every one to
two hours, or more frequently depending on the patient's status and the
physician's orders.
Prompt recognition and intervention are essential.
If you suspect ACS, the physician will need to measure the compartment
pressure by inserting a needle, wick, or slit catheter into the
compartment.
Depending on the reading, a surgeon may need to perform a fasciotomy
—an incision into the fascia of the affected compartment to relieve the
pressure and restore circulation.
Precisely how high a reading merits a fasciotomy is controversial.
07/18/09 24