Escolar Documentos
Profissional Documentos
Cultura Documentos
University Pelita Harapan Faculty of Medicine keep young with charming students
Head Injury
10% of A/E workload Significant cost Expeditious management reduces secondary brain injury Associated injuries and secondary effects High proportion of patients have a subsequent disability
Pathophysiology of TBI
Cerebral Herniation
Subfalcine (Cingulate) Central Uncal (Transtentorial) Tonsillar (Foramen Magnum) Sphenoid (Alar) Transcalvarial Extra Cranial
Pre-Hospital Care
Airway management Transportation Properly trained professionals Prevention of secondary injury
Prehospital Management
Double-convex sign
Epidural Haematoma
Epidural Haematoma
Operation Procedure
Subdural Haemorrhages
Subdural Hemorrhage
Early
Pre Op
Surgery
Post-op
urs
Acci dent
Periorbital bruising Subconjunctival haemorrhage CSF rhino/otorrhoea Epistaxis Haemotympanum Battles sign
HISTORY
Mechanism of Injury (MOI)
Symptoms
LOC Amnesia Nausea and/or vomiting Epistaxis Visual disturbance Headache Dizziness/drowsiness
Motor response
Verbal response
GCS < 15 at any time since the injury Amnesia Neurological symptoms Clinical evidence of a skull fracture Significant extracranial injuries MOI not trivial Continuing uncertainty about diagnosis Medical co-morbidity Adverse social factors
GCS < 15 or GCS 15, but: MOI not trivial LOC Amnesia or has vomited Full thickness scalp laceration/boggy haematoma Inadequate history
Skull X-ray
Advantages
Quick No need for radiologist Low dose of radiation (0.14mSv) Inexpensive
Disadvantages
Increased workload Inconclusive
CT Indications - SIGN
GCS 12/15 or less Deteriorating GCS or progressive focal neurological signs Confusion or drowsiness (GCS 13-14) followed by failure to improve within at most 4 hours of clinical observation Radiological/clinical evidence of fracture GCS 15, no fracture but:
CT Scan
Advantages
High sensitivity/specificity Detection of intracranial haematoma Definitive (except ultra early)
Disadvantages
High dose of radiation (2.0mSv) Radiologist required
EPIDURAL HEMATOMA
Associated with skull fracture. Classic : Middle meningeal artery tear. Lenticular/biconvex due to dural adherence to skull. Lucid interval.
EPIDURAL HEMATOMA
Can be rapidly fatal. Early evacuation good prognosis. Venous epidurals, Possible nonsurgical management.
SUBDURAL HEMATOMA
Venous tear/brain laceration. Covers entire cerebral surface. Morbidity/mortality due to underlying brain injury. Rapid surgical evacuation recommended especially if > 5mm shift of midline.
CONTUSION/HEMATOMA
Coup/contrecoup injuries. Most common : frontal/temporal lobes. Salt & pepper appearance on CT. CT changes usually progressive. Most conscious patients : no operation.
Admission or Discharge?
Continuing amnesia Continuing nausea/vomiting Severe headache Any seizure Focal neurological signs Skull fracture Abnormal CT
None of the above exclusion criteria Patient must be given head injury advice Responsible adult to supervise the patient Easy access to a telephone Reasonable access to a hospital Easy access to transport
Transfer to Neurosurgery
Abnormal CT scan CT is indicated but cannot be done within an appropriate period Clinical features which warrant neurosurgical assessment, monitoring or management:
Persisting coma (GCS 8/15) Persisting confusion Deteriorating GCS Progressive focal neurology Seizure without full recovery Depressed skull fracture Penetrating injury CSF leak/BOS fracture
Medical Management
Intra venous fluids Euvolemia. Isotonic. Hyperventilation, if necessary Goal : PaCO2 at 25 - 35 mmHg.
Medical Management
Mannitol Use with signs of increased ICP Dose : 0,5 - 1,0 g/kg IV bolus. Other Anticonvulsants. Sedation. Paralytics
May be life-threatening if expanding rapidly. Immediate neurosurgical consult. Hyperventilation/Mannitol. ? Emergency burr holes ?
QUESTIONS?