Escolar Documentos
Profissional Documentos
Cultura Documentos
Common problem
High morbidity and
mortality
AN ATO M I
M EN IN G EN
LCS
10mm Hg
= Normal
>20 mm Hg = Abnormal
>40 mm Hg = Severe
Many pathologic processes affect
outcome
ICP Brain function,outcome
M O N RO -KELLIE
C lassif c
iations of H ead injury
Blunt
By
Mechanism
Penetrating
Mild
By
Moderate
Severity
Severe
High velocity
Low velocity
GSW
Other
GCS = 14-15
GCS = 9-13
GCS = 3- 8
C lassif c
iations of H ead Injury
By Morphology
Focal Injury:
Epidural
Subdural
Subarachnoid
Intracerebral
Diffuse Injury
Epidural H em atom a
Subdural H em atom a
D if u
f se A xonal Injury
Frequent reevaluation/ABGs
Circulation
Hypotension not due to brain injury
Hypotension causes secondary
brain injury
Correct hypotension quikly
Do not treat BP, maintain CPP
Disability
GCS
Eye opening
Best motor response
Verbal response
Disability
Minineurologic exam
On patient arrival
After resusciation
Frequently
Document changes
Cause
IIIrd Nerve
compression
bilaterally
Inadequate CNS
perfusion
IIIrd nerve
compression
tentorial
herniation
Cause
Drugs
Pontine lesion
Injured
sympathetic
pathway
H erm ation
Intravenous fluids
Euvolemia
Isotonic
Hyperventilation, if necessary
Goal : PaCO at 25-35 mm Hg
Mannitol
Use with signs of tentorial
herniation
Dose : 0.5 1.0 g/kg IV bolus
Other
Anticonvulsants
Sedation
Paralytics