This document discusses acute coronary syndrome and risk factors for cardiovascular disease. It defines myocardial infarction as myocardial cell death due to prolonged ischemia, noting that cell death occurs histologically after 20 minutes, and complete necrosis of cells occurs within 2-4 hours. It lists modifiable risk factors like smoking, dyslipidemia, raised LDL cholesterol, low HDL cholesterol, raised triglycerides, raised blood pressure, diabetes, obesity, and dietary/lifestyle factors. Non-modifiable risks include personal and family history of coronary artery disease, age, and gender. The document also describes how to assess chest pain characteristics and potential presentations of angina.
This document discusses acute coronary syndrome and risk factors for cardiovascular disease. It defines myocardial infarction as myocardial cell death due to prolonged ischemia, noting that cell death occurs histologically after 20 minutes, and complete necrosis of cells occurs within 2-4 hours. It lists modifiable risk factors like smoking, dyslipidemia, raised LDL cholesterol, low HDL cholesterol, raised triglycerides, raised blood pressure, diabetes, obesity, and dietary/lifestyle factors. Non-modifiable risks include personal and family history of coronary artery disease, age, and gender. The document also describes how to assess chest pain characteristics and potential presentations of angina.
This document discusses acute coronary syndrome and risk factors for cardiovascular disease. It defines myocardial infarction as myocardial cell death due to prolonged ischemia, noting that cell death occurs histologically after 20 minutes, and complete necrosis of cells occurs within 2-4 hours. It lists modifiable risk factors like smoking, dyslipidemia, raised LDL cholesterol, low HDL cholesterol, raised triglycerides, raised blood pressure, diabetes, obesity, and dietary/lifestyle factors. Non-modifiable risks include personal and family history of coronary artery disease, age, and gender. The document also describes how to assess chest pain characteristics and potential presentations of angina.
MI is definined as myocardial cell death due to prolonged ischemia 20 mnt : cell death histologically Several hours : myocardial necrosis 2 4 hours: complete necrosis of myocardial cells Risk Factors for MODIFIABLE NON-MODIFIABLE Cardiovascular PERSONAL SMOKING Disease HISTORY OF CAD DISLIPIDEMIA FAMILY HISTORY OF CAD RAISED LDL AGE CHOLESTEROL GENDER LOW HDL CHOLESTEROL RAISED TRIGLYCERIDE RAISED BLOOD PRESSURE DIABETES MELLITUS OBESITY DIETARY FACTORS THROMBOGENIC FACTORS LACK OF EXERCISE EXCESS ALCOHOL CONSUMPTION Assessing Chest Pain (Classic Angina) LOCATION RADIATION CHARACTER TIME OF ONSET, DURATION, FREQUENCY EXACERBATING AND ALLEVIATING FACTORS ASSOCIATED SYMPTOMS Possible presentation of Angina Angina at rest, with pain episodes lasting > 20 min New onset ( within 2 months )exertional angina of at least CCSC II Recent increase (< 2 months ) in anginal severity to at least CCSC III Angina post MCI Angina Pectoris Stable : there is no substantial deterioration in symptom over several weeks. Stability or quiescence of an atherosclerotic plaque, depending on increased oxygen demand
Unstable : symptom pattern
worsen abruptly without an obvious caused of increased Angina Pectoris
Non Anginal caused of chest pain Musculosceletal disorders GI disorders ( peptic ulcers ) Aortic dissection Thoracic aneurysm Esophageal spasm Pneumonia Pericarditis Neuropsychiatric causes (eg. Panic disorder ) Thank You