Escolar Documentos
Profissional Documentos
Cultura Documentos
3
Disability
Age and LBP:
Leading cause of disability of adults < 45 years old
Third cause of disability in those > 45 years old
Prevalence rate:
Increased 140 % from 1991 to 2000 with only125 % population
growth
Nearly 5 million people in the U.S. are on disability for LBP
4
Lifetime Return to Work
Success of < 50 % if off work > 6 months
5
Differential Diagnoses
Lumbar Strain
Disc Bulge / Protrusion / Extrusion
producing Radiculopathy
Degenerative Disc Disease (DDD)
Spinal Stenosis
Spondyloarthropathy
Spondylosis
Spondylolisthesis
Sacro-iliac Dysfunction
6
“Red Flags” in back pain
Age < 15 or > 50
Fever, chills, UTI
Significant trauma
Unrelenting night pain; pain at rest
Progressive sensory deficit
Neurologic deficits
Saddle-area anesthesia
Urinary and/or fecal incontinence
Major motor weakness
Unexplained weight loss
Hx or suspicion of Cancer
Hx of Osteoporosis
Hx of IV drug use, steroid use, immunosuppression
Failure to improve after 6 weeks conservative tx
Frequency of Back Pain Types
visceral 2%
2% visceral ,tumor
1% 1%
tumor,
,infection
infection,
inflammatory
inflammatory
arthritis
arthritis
97%
“mechanical”
8
Your patient with LBP has paresthesias in the lateral foot, decreased
toe-raise strength, diminished sensation lateral foot, and diminished
Achilles reflex. This is suggestive of dysfunction of which nerve root?
1. L4
2. L5
3. S1
4. S2
Better anatomy knowledge
=
Better diagnoses and treatments
Vertebra
Body, anteriorly
Functions to support weight
Vertebral arch, posteriorly
Formed by two pedicles and two laminae
Functions to protect neural structures
Biomechanics
80% 20%
Anterior Posterior
www.brain101.info 15
Ligaments
Anterior longitudinal ligament
Posterior longitudinal ligament
Ligamentum flavum
Interspinous ligament
Supraspinous ligament
Ligamentous
tt
ee
h
k
bu
c
ig
kn
ba
th
e
e
th
e
th
th
th
in
w
p
o
gu
n
gt
i
el
Pa
t in
t in
gb
a
t in
di
di
ra
ra
a
di
n
ra
i
i
Pa
Pa
ni
Pa
•L4
•L5
•S1
PATIENT HISTORY
“OPQRSTU”
Onset
Palliative/Provocative factors
Quality
Radiation
Severity/Setting in which it occurs
Timing of pain during day
Understanding - how it affects the patient
Which one is NOT considered a
“red flag” of LBP?
1. History/suspicion of cancer 25% 25% 25% 25%
2. Age over 50
3. Fever or chills
4. Sciatica
a
s
50
er
ll
tic
hi
nc
er
ia
rc
ca
ov
Sc
ro
of
ve
Ag
y
or
Fe
st
Hi
“Red Flags” in back pain
Age < 15 or > 50
Fever, chills, UTI
Significant trauma
Unrelenting night pain; pain at rest
Progressive sensory deficit
Neurologic deficits
Saddle-area anesthesia
Urinary and/or fecal incontinence
Major motor weakness
Unexplained weight loss
Hx or suspicion of Cancer
Hx of Osteoporosis
Hx of IV drug use, steroid use, immunosuppression
Failure to improve after 6 weeks conservative tx
Onset
Acute - Lift/twist, fall,
MVA
Subacute - inactivity,
occupational (sitting,
driving, flying)
Pain effect on:
work/occupation
sport/activity (during or
after)
Other History
Prior h/o back pain
Prior treatments and response
Exercise habits
Occupation/recreational
activities
Cough/valsalva exacerbation
Diagnoses & Red Flags
Cancer Fracture
Age > 50 Age >50
History of Cancer Trauma
Weight loss
Steroid use
Unrelenting night pain
Failure to improve Osteoporosis
Inspection
Palpation
Strength testing
Neurologic examination
Special tests
Approach to LBP
History & physical exam
Classify into 1 of 4:
LBP from other serious causes
Cancer, infection, cauda equina, fracture
LBP from radiculopathy or spinal stenosis
Non-specific LBP
Non-back LBP
Workup or treatment
Diagnostic Tools
1. Laboratory:
• Performed primarily to screen for other disease etiologies
Infection
Cancer
Spondyloarthropathies
• No evidence to support value in first 7 weeks unless with red flags
• Specifics:
WBC
ESR or CRP
HLA-B27
Tumor markers: Kidney Breast Lung Thyroid
Prostate
29
2. Radiographs:
30
www.brain101.info 31
3. Electromylogram (EMG):
Measures muscle function
Would not be appropriate in clinically obvious
radiculopathy
4. Bone Scan:
Very sensitive but nonspecific
Useful for:
Malignancy screening
32
33
5. Myelogram:
34
6. CT with Myelogram:
Can demonstrate much better anatomical detail than Myelogram
alone
Utilized for:
Demonstrating anatomical detail in multi-level disease in pre-
operative state
Determining nerve root compression etiology of disc versus
osteophyte
Surgical screening tool if equivocal MRI or CT
35
7. CT:
Best for bony changes of spinal or foraminal stenosis
Also best for bony detail to determine:
Fracture
Malignancy
36
9. MRI
• Best diagnostic tool for:
Soft tissue abnormalities:
• Infection
• Bone marrow changes
• Spinal canal and neural foraminal contents
Emergent screening:
• Cauda equina syndrome
• Spinal cored injury
• Vascular occlusion
• Radiculopathy
Benign vs. malignant compression fractures
Osteomyelitis evaluation
Evaluation with prior spinal surgery
37
Disc Degeneration: Findings?
Narrowing
Osteophyts
Endplate sclerosis
38
Degeneration & Tears
www.brain101.info 39
Disc
Normal Bulge
Bony
Endplate Canal
Disc Classification
Protrusion Extrusion
40
Bulging
www.brain101.info 41
Protrusion
www.brain101.info 42
Protrusion
www.brain101.info 43
Extrusion
www.brain101.info 44
Extrusion
www.brain101.info 45
Extrusion
www.brain101.info 46
Treatment
Pharmacological
NSAIDS
Muscle relaxents:
Re-establish sleep patterns
Membrane stabilizers
TCA / Neurontin
47
Physical Therapy
Modalities
• Electrical Stimulation/TENS
• Postural Education / Body Mechanics
• Massage / Mobilization / Myofascial Release
• Stretching / Body Work
• Exercise / Strengthening
• Traction
• Pre-conditioning / Work-conditioning
48
Epidural Steroid Injections
Indicated for radiculopathy not responding to
conservative mgmt
Conflicting evidence
Small improvement up to 3 months
Less effective in spinal stenosis
Surgery
Laminectomy
Hemilaminectomy
Discectomy
Fusion
Instrumented
Non-instrumented fusion
Minimally Invasive Spine Surgery (MISS)
Kyphoplasty
Percutaneous Disc Decompression (PDD)
www.brain101.info 51
Break for
Physical Examination Hands-on
Session
Inspection
Observe for areas of erythema
Infection
Long-term use of heating element
Unusual skin markings
Café-au-lait spots
Neurofibromatosis
Hairy patches, lipomata
Tethered cord
Dimples, nevi (spina bifida)
Inspection (cont.)
Posture
Shoulders and pelvis should be level
Bony and soft-tissue structures should appear symmetrical
Normal lumbar lordosis
Exaggerated lumbar lordosis is common characteristic of
weakened abdominal wall
Neurologic Examinaion
Includes an exam of entire lower extremity, as lumbar spine
pathology is frequently manifested in extremity as altered
reflexes, sensation and muscle strength
Describes the clinical relationship between various muscles,
reflexes, and sensory areas in the lower extremity and their
particular cord levels
Neurologic Examination
(T12, L1, L2, L3 level)
Motor
Iliopsoas - main flexor of hip
With pt in sitting position, raise thigh against resistance
Reflexes - none
Sensory
Anterior thigh
Neurologic Examination
(L2, L3, L4 level)
Motor
Quadriceps - L2, L3, L4, Femoral Nerve
Hip adductor group - L2, L3, L4, Obturator N.
Reflexes
Patellar - supplied by L2, L3, and L4, although essentially an
L4 reflex and is tested as such
L2, L3, L4 testing
Neurologic Examination
(L4 level)
Motor
Tibialis Anterior
Resisted inversion of ankle
Reflexes
Patellar Reflex (L4)
Sensory
Medial side of leg
Neurologic Examination
(L5 level)
Motor
Extensor Hallicus Longus
Resisted dorsiflexion of great toe
Reflexes - none
Sensory
Dorsum of foot in midline
Neurologic Examination
(S1 level)
Motor
Peroneus Longus and Brevis
Resisted eversion of foot
Reflexes
Achilles
Sensory
Lateral side of foot
Special Tests
Tests to stretch spinal cord or sciatic
nerve
Tests to increase intrathecal pressure
Tests to stress the sacroiliac joint
Tests to Stretch the Spinal Cord or
Sciatic Nerve
Straight Leg Raise
Cross Leg SLR
Kernig Test
Test to increase intrathecal pressure
Valsalva Maneuver
Reproduction of pain
suggestive of lesion
pressing on thecal sac
Kernig Sign
Pain relieved
Pain present
Tests to stress the Sacroiliac Joint
FABER Test
FABER test:
Flexion
A-
Bduction
External
Rotation
Management of an acute low back muscle strain
should consist of all the following EXCEPT:
1. X-rays to rule out a
fracture
2. Educate the patient
on generally good
prognosis
3. Non-opiate analgesics
4. Remain active