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• Tendencies with Pessimism & Optimism can be identified from the Iris & BIP (Iridologia
Moderna - Andrews - published in 2018) - we can work with the Px with these traits as
these traits can negatively and positively influence the PNEI Network and in particular
Natural killer cells, macrophages, neutrophils - numbers & activity
• Koh et al in South Korea looked at the connections with the PNEI Network and immune
cells in autoimmunity, depression and other inflammatory diseases
• Prolactin, Cortisol, Leptin, Growth Hormone CRP & Insulin can all be increased with
Pessimistic behavior and views - they are decreased with Optimistic Px
• Optimistics recover from viruses faster & other diseases as well and have better Zinco
Hipotálamo 4
4
Hipocampo 5
6
Amigdala cerebral
4 Hipotálamo
5 Hipocampo
6 Amigdala cerebral
Aviso: Não concebido como informação ou diagnóstico médico. Apenas para propósitos educativos. Apenas para tendências genéticas.
Hipotálamo 4
4
Hipocampo 5
6
Amigdala cerebral
4 Hipotálamo
5 Hipocampo
6 Amigdala cerebral
Aviso: Não concebido como informação ou diagnóstico médico. Apenas para propósitos educativos. Apenas para tendências genéticas.
Hypothalamus
Hippocampus
Amygdala
Disclaimer: Not intended as a medical diagnosis or information. For educational purposes only. Genetic tendencies only
Amygdala
• 10.1016/j.neuroscience.2015.09.070.
Abstract
Thyroid hormones (THs) play an obligatory role in many fundamental processes underlying brain development and maturation. The
developing embryo/fetus is dependent on maternal supply of TH. The fetal thyroid gland does not commence TH synthesis until mid gestation,
and the adverse consequences of severe maternal TH deficiency on offspring neurodevelopment are well established. Recent evidence
suggests that even more moderate forms of maternal thyroid dysfunction, particularly during early gestation, may have a long-lasting influence
on child cognitive development and risk of neurodevelopmental disorders. Moreover, these observed alterations appear to be largely
irreversible after birth. It is, therefore, important to gain a better understanding of the role of maternal thyroid dysfunction on offspring
neurodevelopment in terms of the nature, magnitude, time-specificity, and context-specificity of its effects. With respect to the issue of context
specificity, it is possible that maternal stress and stress-related biological processes during pregnancy may modulate maternal thyroid
function. The possibility of an interaction between the thyroid and stress systems in the context of fetal brain development has, however, not
been addressed to date. We begin this review with a brief overview of TH biology during pregnancy and a summary of the literature on its
effect on the developing brain. Next, we consider and discuss whether and how processes related to maternal stress and stress biology may
interact with and modify the effects of maternal thyroid function on offspring brain development. We synthesize several research areas and
identify important knowledge gaps that may warrant further study. The scientific and public health relevance of this review relates to achieving
a better understanding of the timing, mechanisms and contexts of thyroid programing of brain development, with implications for early
identification of risk, primary prevention and intervention.
PAX6 gene can be influenced
by nutrition, emotions & stress
Generais:
Stairstep (Reticolo) Lacuna
= Workaholic, “Cannot switch off”,
very motivated/driven
• Indecision
• Challenges/Difficulties with making the right choice
• Inhibition (nutritional absorption)
• Feeling Trapped
• Humiliation/Feeling humiliated
Ileum
• Fatigue
• Neuromuscular tension
• Inflammation
• Insomnia
• Chest pains
• Palpitations/Tachycardia
• Right Iris:
• Cousin/Friend/spouse Conflict
• Sibling Conflict (predominates)
• Left Iris:
• Sibling Conflict (predominates)
• Conflict with Cousin/Friend/Spouse
Descending Colon
• Conflict/stress with:
• Children
• Grandchildren, Godchildren or Students
• Intestinal polyps -crystallisation of
emotional stress & conflict, “energy cyst”
Sigmoid Colon
• Internal Lacuna
• Crypt
• Defect Sign
Th2
Hipotálamo 4
4
Hipocampo 5
6
Amigdala cerebral
4 Hipotálamo
5 Hipocampo
6 Amigdala cerebral
Aviso: Não concebido como informação ou diagnóstico médico. Apenas para propósitos educativos. Apenas para tendências genéticas.
• Developing science
• Recognizes the diverse & profound influence stress has on the individual
• Shows the central importance to all function of the limbic system (emotional
centres of the brain) & the PNEI via the hypothalamus
Symptoms are the tip of the iceberg - the end result of various pathways or
triggers. The iris shows the foundation - the body of the iceberg hidden
under the surface - which contributes to the formation of the tip.
The Collarette is not just the boundary between GI System/ciliary
organs, the internal/external world, but also the potential of the
cell - the extracellular and intracellular environments
Vitex agnus-castus
How Chronobiology can
influence therapeutic approach
• Glutamic acid (which makes GABA) rich foods are: almonds, halibut, mackerel, jumbo oats, beef liver,
walnuts, rice bran, lentils.
www.johnandrewsiridology.net
• Dandruff
• Frequent urination
• Depression
• ADD
• Soft nails
• Allergies
• Lowered immunity
• Weakness
8 or more of these symptoms from Dr Michael
Schmidt’s Livro Smart Fats suggest < EFAs
• Muscle weakness
• Getting out of breath climbing stairs & not linked to respiratory condition
• Fatigue
• Dry hair
• Agressiveness
US $14.95
Can $15.95
UK £8.99
DE S I G N YOUR P E R S ON A L P L A N
HOW CAN WE ENSURE WE LIVE A HEALTHY,
F O R A L O N G A N D H E A LT H Y L I F E
LONG E R
HAPPY AND LONG LIFE? WHAT ARE THE SECRETS
OF AGEING WELL? AND WHY DO SOME PEOPLE AGE
SO MUCH BETTER THAN OTHERS?
LONGE R
we can all take to achieve and maintain optimum health.
younger
prevention of many conditions including dementia and heart disease
yo
An individualized approach to assessing “How Old Is Your Oldest Part”
r
so that you can aim at preventing complications of ageing such as lack
of drive and libido, menopause, arthritis and osteoporosis.
Melatonin
| |
Ser
ine oto
am nin
op
D
Melatonin
|
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Ser
ne oto
mi
|
a
ni
p
n
Do
Acety
|
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GABA
lcholine
Acet
Inne
Inner P llary Borde
r Pupil
GABA
ylcholine
Collarette
lary Border
upi
|
|
|
ine
|
Glut
m
ista
ma a
H
te
e
G
min
luta
|
ta
Nor
m
line
His
adre
ate
a
Adren |
naline
|
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Nor
line na nalin
adre
Adre e
|
Collarette
• Progesterona D6 drops
• > Cortisol - overreaction to stress - “wired” resulting in lower melatonin and brain fog
• Chronic > Cortisol = hippocampal atrophy in the limbic system. > Cortisol = < BDNF
(BDNF D6 drops, progesterone D6, coenzyme q-10,
• Sleep fundamental for melatonin. Gastric ulcers > in blood group A. = Melatonin
required for healthy gut mucus & mucus membranes. Chronobiology studies with
melatonin imbalance showed a connection with gastric inflammation & carcinoma
Principle Neurotransmitters
• Adrenaline (A) is a stress hormone and promotes the release of glucose from the liver. It is produced in the adrenal glands
and the brain. Long-term excess may result in anxiety, cardiovascular problems an accumulation of abnormal cells and
problems with glucose utilisation. Long-term deficiency can be associated with fatigue, depression, an accumulation of
abnormal cells and digestive problems
• Noradrenaline (NA) is one of the major neurotransmitters with many functions. It is produced in the brain ans the adrenal
glands. Long-term excess may result in increased blood-pressure, inflammation, hyperthyroidism and panic attacks. Long-
term deficiency may result in impaired thought processes, behavioural problems, immunological imbalance, hypothyroidism
and the development of various pathological conditions
• Dopamine (DA) is one of the major neurotransmitters. It is produced in the brain, the kidneys and the adrenal glands. Long-
term excess is very often associated with over activation of the immune system, inflammation and hormonal imbalance, whilst
long-term deficiency may result in the impairment of some movement, memory problems, mood swings, immunological and
hormonal imbalances, kidney problems and also the development of various pathological conditions
• Serotonin (5-HT) is one of the major neurotransmitters. It is produced in the brain and the gut. Long-term excess may result
in cardiovascular problems, hormonal and immunological imbalances and behavioural problems, whilst long-term deficiency
could be associated with depression, stress, insomnia, inflammation, gut problems, hormonal and immunological problems
and also the development of various pathological conditions
• γ-Aminobutyric Acid (GABA) is a major inhibitory neurotransmitter. It is produced in the brain. Long-term excess may result
in memory problems, impairment of movement, problems with breathing, immunological imbalance and the development of various pathological
conditions. Long-term deficiency is very often associated with anxiety, memory problems, insomnia, behavioural problems
and an immunological imbalance
• Acetylcholine (ACH) is one of the major neurotransmitters and has
been shown to be essential for brain development. Long-term excess
may be associated with the exhaustion of pancreatic activity,
inflammation, auto-immunity, behavioural problems and the
development of various pathological conditions
• Excitatory neurotransmitters stimulate the brain. Those that calm the brain and
help create balance are called inhibitory. Inhibitory neurotransmitters balance
mood and are easily depleted when the excitatory neurotransmitters are
overactive
• The brain uses neurotransmitters to tell your heart to beat, your lungs to breathe,
and your stomach to digest. They can also affect mood, sleep, concentration,
weight, and can cause adverse PNEI symptoms when they are out of balance
• From 1,193 iridology Px between 1999 to 2014 - 738 Px had NT issues &
tendencies with tyrosine (dopamine, adrenaline, noradrenaline) tryptophan
(serotonin/melatonin), histamine, GABA & acetylcholine (choline)
Melatonin
| |
Ser
ine oto
am nin
op
D
Melatonin
|
| |
Ser
ne oto
mi
|
a
ni
p
n
Do
Acety
|
|
GABA
lcholine
Acet
Inne
Inner P llary Borde
r Pupil
GABA
ylcholine
Collarette
lary Border
upi
|
|
|
ine
|
Glut
m
ista
ma a
H
te
e
G
min
luta
|
ta
Nor
m
line
His
adre
ate
a
Adren |
naline
|
|
Nor
line na nalin
adre
Adre e
|
Collarette
• 229 Male
• Iris & IPB Signs were correlated with the Px’s test results
when possible. The tests consisted of 571 urine tests, 247
saliva tests & 2 blood tests
Melatonin
| |
Ser
ine oto
am nin
op
D
Melatonin
|
| |
Ser
ne oto
mi
|
a
ni
p
n
Do
Acety
|
|
GABA
lcholine
Acet
Inne
Inner P llary Borde
r Pupil
GABA
ylcholine
Collarette
lary Border
upi
|
|
|
ine
|
Glut
m
ista
ma a
H
te
e
G
min
luta
|
ta
Nor
m
line
His
adre
ate
a
Adren |
naline
|
|
Nor
line na nalin
adre
Adre e
|
Collarette
• Small does of single substance stimulate, moderate doses of the same substance
can inhibit and large doses can stop physiological processes (or even kill, which is
not the desired aim of homotoxicology!))
• Research: studies with Arnica, Nux Vomica, Cortisone & others - the higher the
dilution of the substance the greater & safer the therapeutic action, the higher the
crude dose the more toxic and pro-inflammatory the results
• Magnesium is vital to all cellular functions and carrying many substances into the cell -
one of the foundations to most biochemical activity in the body is sufficient and
efficient magnesium levels
• Malic acid is essential for muscular functions, good energy levels, mental clarity,
mucosal integrity & correct immune response - so we can see how a lower level or
disruption of certain arms within the CAC can trigger or maintain chronic fatigue or
fibromyalgia conditions
FIBROBLASTS
• Autoimmune diseases are on the rise across the globe
including UK, Denmark, Italy, Ireland, France, USA, Japan &
Brasil
• UC - Cytokines
• UC - Emotional Dynamics - Fleche
• UC - Emotional Dynamics - Andrews
• UC - Autoimmunity
• UC - Neurotransmitters
• Evid Based Complement Alternat Med. 2016;2016:9872598. Epub 2016 Oct 27.
Henriques BO1, Corrêa O1, Azevedo EP1, Pádua RM1, de Oliveira VL2, Oliveira TH2, Boff D2, Dias AC3, de Souza DG3, Amaral FA2, Teixeira MM2, Castilho RO1, Braga FC1.
Abstract
Stryphnodendron species, popularly named "barbatimão," are traditionally used in Brazil as anti-inflammatory agents. This study aimed to investigate the effect of barbatimão and 11 other species on
the production of tumor necrosis factor-alpha (TNF-α) in lipopolysaccharide- (LPS-) stimulated THP-1 cells, as well as their anti-arthritis activity. The extracts of Stryphnodendron
adstringens, Stryphnodendron obovatum, Campomanesia lineatifolia, and Terminalia glabrescens promoted a concentration-dependent inhibition of TNF-α. Mice injected with LPS in the knee joint were
treated per os with fractions from the selected extracts. Both the organic (SAO) and the aqueous (SAA) fractions of S. adstringens promoted a dose-dependent reduction of leukocyte migration and
neutrophil accumulation into the joint, but none of them reduced CXCL1 concentration in the periarticular tissue. In contrast, treatment with C. lineatifolia and T. glabrescens fractions did not ameliorate
the inflammatory parameters. Analyses of SAO by Ultra Performance Liquid Chromatography (UPLC) coupled to electrospray ionization mass spectrometry (ESI-MS) led to the identification of gallic
acid along with 11 prodelphinidins, characterized as monomers and dimers of the B-type. Our findings contribute to some extent to corroborating the traditional use of S. adstringens as an anti-
inflammatory agent. This activity is probably related to a decrease of leukocyte migration into the inflammatory site. Polyphenols like gallic acid and prodelphinidins, identified in the active fraction, may
contribute to the observed activity.
Evid Based Complement Alternat Med. 2015;2015:497123. doi: 10.1155/2015/497123. Epub 2015 Mar 23.
Evaluation of the Effects of Some Brazilian Medicinal Plants on the Production of TNF- α and CCL2 by THP-1 Cells.
Abstract
Several plant species are traditionally used in Brazil to treat various inflammatory diseases. Tumor necrosis factor- (TNF-) α and chemokine (C-C motif) ligand 2 (CCL2) are key inflammatory mediators
in diseases like rheumatoid arthritis and atherosclerosis, respectively; nevertheless, only a few extracts have been assayed against these targets. We herein report the effect of 19 plant extracts on TNF-
α and CCL2 release by lipopolysaccharide- (LPS-) stimulated THP-1 cells, a human monocytic leukemia cell line, along with their radical scavenging activity on DPPH. The extracts of Caryocar
brasiliense, Casearia sylvestris, Coccoloba cereifera, and Terminalia glabrescens inhibited TNF-α production in a concentration-dependent manner. Fractionation of these extracts potentiated the anti-
TNF-α effect, which was shown to concentrate in polar fractions, mainly composed by polyphenols. Significant CCL2 inhibition was elicited by Lippia sidoides and Terminalia glabrescens extracts,
whose fractionation resulted in highly active low polar fractions. All assayed extracts showed strong radical scavenging activity, but antioxidant activity did not correlate with inhibition of TNF-α or CCL2
production. Our results allowed identifying extracts with selective capacity to block cytokine production; therefore, further purification of these extracts may yield molecules that could be useful in the
treatment of chronic inflammatory diseases.
• Trends Immunol. 2006 Sep;27(9):399-404. Epub 2006 Jul 13.
Abstract
For several years, reproductive immunology has been dominated by the 'Th1/Th2' hypothesis, in which the fetus avoids
maternal T-cell rejection through a bias towards T-helper (Th)2 cytokine production. The discovery that normal pregnancy
is a controlled state of inflammation, at an early stage at the implantation site and also later systemically, has challenged
this concept, as has the finding that the predominant immune interactions in the decidua are between the placental
trophoblast and maternal natural killer (NK) cells instead of T cells. Here, we extend this concept to the interaction
between the trophoblast and NK cells in the maternal circulation. We suggest novel ways in which the trophoblast might
stimulate the maternal systemic inflammatory response, and how dysfunctional NK-cell activation could result in the
maternal syndrome of pre-eclampsia.
LEGENDA
0 - 1 Amígdala Cerebral, Depressão, Suprarrenais 3 - 4 Pâncreas, Disglicemia 6 - 7 Sono, Glândula Pineal, Melatonina
1 - 2 Coração, Tireoide, Hipocampo, Hipotálamo 4 - 5 Progesterona, Mucosa Intestinal, SOP, Testosterona 7 - 8 Estômago
2 - 3 Fígado, Tireoide, Ouvido 5 - 6 Progesterona, Útero, Endometriose 8 - 9 Suprarrenais, Alergias, Asma, Fadiga Crônica
Sinais na íris: * Reentrância do colarete em direção à pupila * Pigmento da Íris * Sulco Radial * Lacuna * Cripta * Aberturas no Colarete
Aviso: Não concebido como informação ou diagnóstico médico. Apenas para propósitos educativos. Apenas para tendências genéticas.
• Prenatal stress & the programming of the HPA Axis, Glover et al, Neuroscience &
Biobehavioral Reviews 35 (2010)17-22
• Prenatal exposure to maternal psychosocial stress & HPA axid regulation in young
adults, Entringer et al. Horm Behav.55. 292-298
• Prenatal trauma & risk for chronic fatigue syndrome - association with neuroendocrine
dysfunction, Heim et al. Arch.Gen. Psychiatry 66, 72-80
• Exposure to stress hormones in the womb linked to adult mood disorders, Wellcome
TrustBlog, 2016 The uterine space is not just a physical space during our gestation
• Diabetes mellitus
• PCOS
• Endometriosis
• Crohn’s disease
• Depression
• Heart Disease
Hipotálamo
Hipocampo
Amigdala cerebral
Aviso: Não concebido como informação ou diagnóstico médico. Apenas para propósitos educativos. Apenas para tendências genéticas.
• Liver - is not necessarily fully engaging with the problem head on, but finding motivation with different solutions,
an all or nothing personality
• Miosis - tend to be hypoactive in mind & action, nervous strong, individual, can be distrusting, love winning &
setting goals & plans, directed towards the material - social success etc in a couple two mitotic px can be too
competitive with each other, need to compare, be creative & fight
• Mydriasis - "asthenic", adrenal fatigue, very open, can be artistic, creative & childlike - looking at life & the world
in awe. melancholy, apathetic, too pen, two mydriatic people or a couple are usually challenged in getting
organised, practicals, feed on insights & beauty - and, palls, medo, very helpful, compliant, sweet disposition, they are
proposers
A traumatic birth experience can result in shock & a fear in the early, - panic &
anxiety with hyperadrenal function in later life - allergies, anxiety, IBS, asthma,
chronic fatigue, food sensitivities, depression, TMJ Syndrome & insomnia can all
result
With this the amygdala is imprinted (Odent), adrenaline, activation of HPA Axis, >
histamine, > PRL more likely in later life, < dopamine,
The prenatal in utero experience for the Px goes anti- clockwise along the collarette
- as placed by Lo Rito
The grandparent and the grandchild’s life and prenatal experience follow the same
direction for the Px
The parents and children follow a clockwise direction in the above experience
PARADIGM
SHIFT
© Dipartimento Scientifico Guna S.p.a.
LOW
DOSE
MEDICINE
© Dipartimento Scientifico Guna S.p.a.
© Dipartimento Scientifico Guna S.p.a.
A HIERARQUIA E DESENVOLVIMENTO GESTACIONAL
DO SISTÊMA LÍMBICO
Hipotálamo 4
4
Hipocampo 5
6
Amigdala cerebral
4 Hipotálamo
5 Hipocampo
6 Amigdala cerebral
Aviso: Não concebido como informação ou diagnóstico médico. Apenas para propósitos educativos. Apenas para tendências genéticas.